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• W1485076888 abstract "1.1 Inflammation Inflammation is a response of the organism that facilitates the immune response and/or repair after a harmful stimulus, such as pathogens, damaged cells, or irritants. The inflammatory process is necessary for the healing of the tissues. However, the uncontrolled chronic inflammation can also lead to harmful diseases, such as atherosclerosis or rheumatoid arthritis, among others (Eming et al., 2007; Tabas, 2010; Libby, 2008). The cause of inflammation may be of different origin. An inflammatory response could be stimulated by chemical or physical damages, but also by pathogens or immune reactions (Moore et al., 2010). The cardinal signs of inflammation in the majority of the tissues include pain, redness, immobility, swelling and heat. These signs usually encompass the increase of blood flow in the inflamed area and the accumulation of fluids that help to increase the possibilities of tissue repair. However, inflammation of particular tissues, such as in the central nervous system (CNS), those characteristics are not fully applicable (Carson et al., 2006). Many cells are implicated in the inflammatory process. One of the initial players is the resident macrophages, that are able to respond rapidly to damage. Particularly in the CNS, the macrophage-analogue cells are the resident microglia but their role is not fully understood and it is some how different from macrophages (Schwartz, 2003). Other tissue resident cells, like Dendritic cells or Kupfer cells, not present in the CNS, may also be initial responders to inflammation but other cells, entering in the CNS and coming from the systemic circulation, such as natural killer (NK) cells, leukocytes, and lymphocytes are also implicated in the inflammatory response (McMahon et al., 2006; Haskill et al., 1992). Inflammation also comprises a complex biological response implicating vascular tissues and the activation of the endothelium to facilitate the specific entrance of blood cells (Trepels et al., 2006). After a specific insult, several cascades of factors are activated. The plasma derived inflammatory mediators such as the complement and the coagulation system, together with" @default.
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• W1485076888 date "2011-10-10" @default.
• W1485076888 modified "2023-10-03" @default.
• W1485076888 title "Acute Phase Protein’s Levels as Potential Biomarkers for Early Diagnosis of Neurodegenerative Diseases" @default.
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• W1485076888 doi "https://doi.org/10.5772/22626" @default.
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