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- W1487881813 abstract "In recent years, research on the genetic basis of inherited cardiac channelopathies, namely the Long QT syndrome, the Brugada syndrome, and more recently catecholamine-induced polymorphic ventricular tachycardia, has initiated an increased understanding of the molecular blueprints of myocardial electrical function. The electrophysiological analysis of naturally-occurring mutant ion channels associated with these disorders provides insight, not only into mechanisms of disease but also, for instance, into the biophysical properties of the native channel, its pharmacology, its association with other subunits and its contribution to the myocardial action potential. This, in turn, should ultimately enable more specific pharmacological intervention in the management of these syndromes and other related and possibly more common arrhythmias." @default.
- W1487881813 created "2016-06-24" @default.
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- W1487881813 date "2003-01-01" @default.
- W1487881813 modified "2023-10-18" @default.
- W1487881813 title "Ion Channel Disease as a Cause of the Brugada Syndrome" @default.
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- W1487881813 doi "https://doi.org/10.1007/978-1-59259-362-0_10" @default.
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