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- W1493303306 abstract "Cumulative evidence suggests that the origins of obesity may occur during fetal development. In this respect, the concept of developmental programming was introduced and supported by experimental and epidemiological data. This concept supports the idea that the nutritional and hormonal status during pregnancy could irreversibly interfere in metabolism control. The mechanisms responsible for this developmental programming remain poorly documented. However, recent research indicates that adipocytokines may play a critical role in this process. Thus, leptin, adiponectin, and the recently identified resistin, visfatin, and apelin, all exert effects on fat, muscle, and liver cells early in life. The aforementioned adipocytokines are secreted by adipocytes and human placenta during fetal life and may play a major role in the etiopathogenesis of the metabolic syndrome. This review will focus on the intrauterine expression of adipocytokines, their contribution to the hormonal control of fetal growth, and their role in restricted and exaggerated intrauterine growth." @default.
- W1493303306 created "2016-06-24" @default.
- W1493303306 creator A5031246847 @default.
- W1493303306 creator A5071664474 @default.
- W1493303306 date "2010-09-01" @default.
- W1493303306 modified "2023-09-23" @default.
- W1493303306 title "The role of adipocytokines in fetal growth" @default.
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- W1493303306 doi "https://doi.org/10.1111/j.1749-6632.2010.05650.x" @default.
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