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• W1497473887 startingPage "153" @default.
• W1497473887 abstract "Clinical and experimental observations on the patterns of metastatic targeting indicate that the involvement of secondary organ sites does not occur at random, but rather, is governed by a large number of possible mechanisms. In one sense, metastasis can be viewed as a normal phenomenon, governed by events and processes that are common to fetal development, inflammation, and wound repair. Thus, the properties of cancer cells that determine their ability to invade local tissues and to colonize distant organs (the so-called “metastatic phenotype”) are also the properties of embryonic cells and of cells responsible for immune processes, inflammation, and tissue healing. Such properties include the ability to adhere to other cells and extracellular matrices, the production of proteolytic enzymes, and cell migration. The subject of this chapter evolved conceptually from observations in vivo that cancer cells have ameboid properties and move by pseudopodial extension. Cancer cell motility can be studied in monolayer cultures and in cell or tissue explants where migration is generally random in terms of direction. From observations of this type, direct correlations have been made between the rates of random cell motility and metastatic behavior in some cell lines." @default.
• W1497473887 created "2016-06-24" @default.
• W1497473887 creator A5049622220 @default.
• W1497473887 date "1994-01-01" @default.
• W1497473887 modified "2023-09-23" @default.
• W1497473887 title "Cancer Cell Chemotaxis: Mechanisms and Influence on Site-Specific Tumor Metastasis" @default.
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