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- W150039393 abstract "The importance of cellular immunity in the host’s response to varicella-zoster virus (VZV) is manifested clinically by the severity of varicella in children with congenital or acquired T-cell deficiencies (1). The frequent occurrence of herpes zoster with suppression of cellular immunity implies that such immunity is also required to prevent symptoms due to viral reactivation. When a VZV antigen preparation made from sonicated infected cells was used to stimulate T lymphocytes, the early acquisition of VZV-specific T-cell proliferation was found to correlate with mild primary VZV infection, the absence of VZV cellular immunity was associated with progressive varicella, and diminished VZV T-cell immunity defined periods of susceptibility to recurrent VZV in patients with malignancy (2–5). In contrast, immunoglobulin deficiencies have not been associated with severe primary VZV disease, and VZV IgG antibody titers are not usually low at the onset of herpes zoster." @default.
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- W150039393 date "1990-01-01" @default.
- W150039393 modified "2023-10-18" @default.
- W150039393 title "The T-Lymphocyte Response to Varicella-Zoster Viral Proteins" @default.
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- W150039393 doi "https://doi.org/10.1007/978-1-4684-5853-4_8" @default.
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