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- W150560933 abstract "Publisher Summary Vitamin A deficiency results in abnormal cell growth and differentiation, and its derivatives—retinol or retinoic acid—restore normal function in most tissues. The ability of retinoic acid to regulate the expression of a variety of protooncogenes such as c-myc, c-myb, and c-jun suggests that these transcription factors are coupled to the regulatory pathways involved in retinoid action. Myb—the nuclear protein product of c-myb—is a high-affinity DNA-binding protein that exhibits both transactivator and transrepressor activity associated with cell growth and differentiation. This chapter describes the transient up-regulation of c-myb mRNA in the rat Sertoli cell in response to vitamin A stimulation. The significant rise in the message for the transcription factor suggests that the nuclear protein Myb may mediate retinol-inducible gene expression. Myb can initiate regulatory changes in the genetic program of the Sertoli cell via transcriptional control. It is demonstrated in this chapter that, the c-myb protooncogene is expressed in cultured rat Sertoli cells and that its expression is stimulated by both retinol and retinoic acid." @default.
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- W150560933 date "1995-01-01" @default.
- W150560933 modified "2023-09-27" @default.
- W150560933 title "Effects of Retinoids on Expression of the Protooncogene c-myb in Rat Sertoli Cells" @default.
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- W150560933 doi "https://doi.org/10.1016/b978-0-12-571150-0.50038-x" @default.
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