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- W150642103 endingPage "242" @default.
- W150642103 startingPage "223" @default.
- W150642103 abstract "Recent advancements in stem cell biology have revealed remarkable plasticity in cell fate specification. For example, fully differentiated somatic cells can be reprogrammed to pluripotent stem cells following the expression of specific stem cell associated transcription factors. This extraordinary process of cellular reprogramming or dedifferentiation shares many similarities with tumor progression, such that cancer cells often acquire stem cell-like plasticity concomitant with metastatic disease. Evidence suggests that cancer cells co-opt stem cell associated signaling factors to sustain plasticity. However, in contrast to normal stem cells, which have a complement of inhibitors and activators of pluripotency, cancer cells lack this critical balance. Here, we describe stem cell associated proteins and microenvironments that sustain and promote cellular plasticity in embryonic and neoplastic populations. We also review evidence that embryonic microenvironments may be capitalized upon to rebalance cancer cells toward a benign well-differentiated phenotype." @default.
- W150642103 created "2016-06-24" @default.
- W150642103 creator A5009027980 @default.
- W150642103 creator A5009869289 @default.
- W150642103 creator A5034140018 @default.
- W150642103 creator A5063543170 @default.
- W150642103 date "2011-01-01" @default.
- W150642103 modified "2023-09-23" @default.
- W150642103 title "Influence of the Embryonic Microenvironment on Tumor Progression" @default.
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