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- W150726061 endingPage "420" @default.
- W150726061 startingPage "395" @default.
- W150726061 abstract "Osteoporosis and other drug-induced metabolic bone diseases develop insidiously but extract a significant public health cost. Uncoupling of bone resorption and formation phases of the bone remodeling cycle contribute to net loss of bone mineral content, microarchitectural derangement, and fracture. Osteoporosis is well documented with use of glucocorticoids, L-thyroxine, heparin, ethanol, caffeine, tobacco, GnRH analogues, neuroleptics, and immunosuppressive agents. Osteomalacia, hypersteroidosis, mineralization defects, and adynamic bone disease are manifest with use of anticonvulsants, biphosphonates, fluoride, and aluminum related to renal osteodystrophy. Recognition of iatrogenic bone disease allows for monitoring of drug use and appropriate preventive interventions." @default.
- W150726061 created "2016-06-24" @default.
- W150726061 creator A5021593867 @default.
- W150726061 date "1995-06-01" @default.
- W150726061 modified "2023-10-18" @default.
- W150726061 title "Drug-Induced Metabolic Bone Disease" @default.
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