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- W1508130697 abstract "It has been suggested that stillbirths are part of the spectrum of infant deaths that includes sudden infant death syndrome (SIDS). This paper examines the hypothesis that risk factors associated with stillbirths might contribute to dysregulation of inflammatory responses to infections that could trigger the physiological responses leading to fetal loss. These include genetic factors (ethnic group, sex), environmental (infection, cigarette smoke, obesity), and developmental (testosterone levels) factors. Interactions between the genetic, environmental, and developmental risk factors are also considered, e.g., the excess of male stillborn infants in relation to the effects of testosterone levels during development on pro-inflammatory responses. In contrast to SIDS, inflammatory responses of both mother and fetus need to be considered. Approaches for examining the hypothesis are proposed." @default.
- W1508130697 created "2016-06-24" @default.
- W1508130697 creator A5062888526 @default.
- W1508130697 date "2015-06-09" @default.
- W1508130697 modified "2023-09-27" @default.
- W1508130697 title "The Role of Infection and Inflammation in Stillbirths: Parallels with SIDS?" @default.
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- W1508130697 doi "https://doi.org/10.3389/fimmu.2015.00248" @default.
- W1508130697 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4460799" @default.
- W1508130697 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26106385" @default.
- W1508130697 hasPublicationYear "2015" @default.
- W1508130697 type Work @default.