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- W1512296869 abstract "Viral infections in neonates and children range from asymptomatic carrier status to end-stage acute or chronic liver failure. Neonates are usually infected in utero or perinatally with the TORCH infections and could develop acute liver failure due to herpes simplex viruses, enteroviruses, or rarely adenoviruses. Acute hepatitis in older children is due to hepatitis A virus (HAV), hepatitis E virus (HEV), or Epstein–Barr virus (EBV) in adolescents. Hepatitis B may present with fulminant hepatitis in neonates of HBe antigen–negative mothers as well as in older children. Acute hepatitis from any source may progress to fulminant hepatitis requiring liver transplantation. Chronic hepatitis in childhood is due to either HBV or HCV. The main route of infection is perinatal, and children have a low natural seroconversion rate and a lifetime risk of chronic liver disease and liver cancer. Treatment for HBV is unsatisfactory, with only a 30% success rate with either interferon or nucleotides, while combination therapy with pegylated interferon and ribavirin clears between 70–90% of HCV infections dependent on the genotype." @default.
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- W1512296869 date "2013-07-26" @default.
- W1512296869 modified "2023-10-14" @default.
- W1512296869 title "Neonatal and Pediatric Infection" @default.
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- W1512296869 doi "https://doi.org/10.1002/9781118637272.ch39" @default.
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