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- W1515691643 endingPage "45" @default.
- W1515691643 startingPage "433" @default.
- W1515691643 abstract "The nuclear accumulation and transcriptional activity of NFκB are constitutively increased in cutaneous T-cell lymphoma (CTCL) cells, and are responsible for their increased survival and proliferation. However, in addition to the anti-apoptotic and pro-inflammatory genes, NFκB induces expression of immunosuppressive genes, such as IL-10 and TGFβ, which inhibit the immune responses and are characteristic for the advanced stages of CTCL. While the mechanisms regulating NFκB-dependent transcription of anti-apoptotic and pro-inflammatory genes have been studied extensively, very little is known about the NFκB regulation of immunosuppressive genes. The specificity of NFκB-regulated responses is determined by the subunit composition of NFκB complexes recruited to the individual promoters, post-translational modifications of NFκB proteins, as well as by their interactions with other transcriptional factors and regulators. In this review, we discuss the mechanisms regulating the transcription of NFκB-dependent anti-apoptotic, pro-inflammatory and immunosuppressive genes in CTCL cells, as potential targets for CTCL therapies." @default.
- W1515691643 created "2016-06-24" @default.
- W1515691643 creator A5042921227 @default.
- W1515691643 creator A5054346929 @default.
- W1515691643 date "2013-11-01" @default.
- W1515691643 modified "2023-09-23" @default.
- W1515691643 title "NFκB function and regulation in cutaneous T-cell lymphoma." @default.
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