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- W1520255523 abstract "Abstract Nonobese diabetic (NOD) mice carrying a segment of chromosome flanking the disrupted IFN-γ receptor gene from original 129 ES cells are resistant to development of diabetes. However, extended backcrossing of this mouse line to the NOD mouse resulted in a segregation of the IFN-γR-deficient genotype from the diabetes-resistant phenotype. These results indicate that the protection of NOD mice from the development of diabetes is not directly linked to the defective IFN-γ receptor gene but, rather, is influenced by the presence of a diabetes-resistant gene(s) closely linked to the IFN-γR loci derived from the 129 mouse strain." @default.
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- W1520255523 date "2000-04-01" @default.
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- W1520255523 title "Protection of Nonobese Diabetic Mice from Diabetes by Gene(s) Closely Linked to IFN-γ Receptor Loci" @default.
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- W1520255523 doi "https://doi.org/10.4049/jimmunol.164.7.3919" @default.
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