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- W1520693724 endingPage "49" @default.
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- W1520693724 abstract "The endothelin (ET) system comprises a family of three isopeptides (ET-1, ET-2, and ET-3)involved in diverse physiological and pathophysiological events. ET-1 is the major renal peptide that exerts its biological activity by binding to ETA and ETB receptors. Both ETA and ETB receptors are expressed by renal microvascular smooth muscle cells, where activation causes vasoconstriction. ETB receptors are also expressed by microvascular endothelial cells, where activation leads to vasodilator responses. ET-1 influences preglomerular and postglomerular microvascular tone and thus can significantly influence renal hemodynamics. Alteration of renal ET-1 synthesis and receptor expression has been reported in cardiovascular diseases, and could contribute to renal injury by altering renal microvascular reactivity. In this brief review, we will try to summarize what is known about ET control of renal microvascular function." @default.
- W1520693724 created "2016-06-24" @default.
- W1520693724 creator A5009600813 @default.
- W1520693724 creator A5063490518 @default.
- W1520693724 date "2011-01-01" @default.
- W1520693724 modified "2023-09-23" @default.
- W1520693724 title "Endothelin and the Renal Vasculature" @default.
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- W1520693724 doi "https://doi.org/10.1159/000328720" @default.
- W1520693724 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3236641" @default.
- W1520693724 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21893987" @default.
- W1520693724 hasPublicationYear "2011" @default.
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