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• W1520705364 abstract "The vasculitides include a broad spectrum of disorders that span a clinical spectrum from benign, self-limited disease to fulminant conditions that are fatal in the absence of therapy. Whereas the large-vessel vasculitides consist of 2 principal disorders, giant cell arteritis and Takayasu arteritis, the mediumand small-vessel vasculitides are much more diverse, including multiple diseases that can affect nearly every organ system (Seo & Stone, 2007). This article is the second of a 2-part series that focuses on the challenges faced by clinicians who care for patients with vasculitis. The first article in this series discussed the large-vessel vasculitides. The problem of systemic autoimmune diseases such as systemic vasculitis with vascular lesions of medium and small vessels, including: Wegener granulomatosis, ChurgStrauss syndrome, microscopic poliangiitis, nodose poliarteriitis, attracts every year more attention of doctors, due to the worldwide relentless increase of patients with these diseases (Lhote & Guillevin, 2009; Tsukadaira, 2009). Recently, several reports have suggested that vasculitis is becoming more common. Systemic vasculitis is a group of heterogeneous diseases (syndromes), characterized by inflammation and damage to blood vessels and which compromises or destroys the vessel wall leading to haemorrhagic and ⁄ or ischaemic events, giving impetus to the development of a wide spectrum of symptoms and signs. The forms of vasculitis may be varied: primary [idiopathic, e.g. cutaneous leukocytoclastic angiitis, Wegener’s granulomatosis, Churg– Strauss syndrome and microscopic polyangiitis ] and secondary [a manifestation of connective tissue disease, infection, adverse drug eruption, or a paraneoplastic phenomenon], local and generalized, transient, recurrent and chronic; included group of diseases in which vasculitis is a primary characteristic. They are general in nature, though sometimes a local clinic can manifest symptoms. General damage of blood vessels may occasionally appear and disappear, but in most cases there are long-term. Each new escalation leads to the increase of clinical symptoms. Clinical implications of SV course depend on a number of factors, including etiology, location, size and number of blood vessels, the extent and severity of disease, its activity and nature of treatment. Systemic vasculitis as the existing disease was first described by A. Cussmaul and I. Maier, when they reported a case of necrotizing arteritis and called it nodose periarteriitis. There are over twenty different forms of Systemic vasculitis. One of the first classification was their classification by P. Zeek's, which included five categories of Systemic vasculitis: nodose" @default.
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• W1520705364 date "2011-10-17" @default.
• W1520705364 modified "2023-10-03" @default.
• W1520705364 title "Churg-Strauss Syndrome: Clinical and Immunological Features" @default.
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• W1520705364 doi "https://doi.org/10.5772/21504" @default.
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