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- W1520784851 abstract "C/EBP homologous protein, an important transcription factor during endoplasmic reticulum stress, participates in cell apoptosis mediated by endoplasmic reticulum stress. Previous studies have shown that C/EBP homologous protein mediates nerve injury during Alzheimer's disease, subarachnoid hemorrhage and spinal cord trauma. In this study, we introduced C/EBP homologous protein short hairpin RNA into the brains of ischemia/reperfusion rat models via injection of lentiviral vector through the left lateral ventricle. Silencing C/EBP homologous protein gene expression significantly reduced cerebral infarction volume, decreased water content and tumor necrosis factor-α and interleukin-1β mRNA expression in brain tissues following infarction, diminished the number of TUNEL-positive cells in the infarct region, decreased caspase-3 protein content and increased Bcl-2 protein content. These results suggest that silencing C/EBP homologous protein lessens cell apoptosis and inflammatory reactions, thereby protecting nerves." @default.
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- W1520784851 date "2012-11-05" @default.
- W1520784851 modified "2023-09-23" @default.
- W1520784851 title "Silencing the gene encoding C/EBP homologous protein lessens acute brain injury following ischemia/reperfusion." @default.
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- W1520784851 doi "https://doi.org/10.3969/j.issn.1673-5374.2012.31.004" @default.
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