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- W1526706844 abstract "Abstract : Alternative lengthening of telomeres (ALT) is a mechanism utilized by several cancer types to maintain telomeres. The aim of this project is to characterize the mechanisms that mediate and regulate ALT activity in breast cancer, by utilizing proteomic and genomic approaches to describe and perturb ALT+ cells. Gaining this knowledge on ALT activity can promote the development of novel treatment for ALT+ breast tumors, and patients with telomerase inhibitor resistant tumors, while having minimal non-desired effects on the normal cell population that typically has no ALT activity. Here, I report on my progress in profiling the global transcriptional patterns that differentially characterize ALT+ cells. I describe the analytical tools that I used to define an ALT-related gene signature, and my efforts to identify potential functional relationship to ALT activity." @default.
- W1526706844 created "2016-06-24" @default.
- W1526706844 creator A5037910480 @default.
- W1526706844 date "2013-04-01" @default.
- W1526706844 modified "2023-09-24" @default.
- W1526706844 title "Systematic Characterization of the Molecular Mechanisms That Regulate and Mediate Alternative Lengthening of Telomeres in Breast Carcinoma" @default.
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- W1526706844 doi "https://doi.org/10.21236/ada581164" @default.
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