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- W1539928018 abstract "Senescence is a state of permanent proliferation arrest that normal cells undergo in response to shortened telomeres, oncogenic activation and other sources of cellular stress, thus restricting the replicative capacity of impaired or damaged cells. As such, senescence provides a potent mechanism of tumor suppression, but has also been implicated in organismal aging. Senescence is accompanied by profound chromatin remodeling, which reinforces several important features of the senescence program. Consequently, there is considerable interest in elucidating precisely how chromatin structure influences senescence. In the present work, I set out to investigate the role of the H4K20me3 histone modification in senescence, as the mark has been implicated in aging and is commonly lost in human cancers." @default.
- W1539928018 created "2016-06-24" @default.
- W1539928018 creator A5079961444 @default.
- W1539928018 date "2012-01-01" @default.
- W1539928018 modified "2023-09-23" @default.
- W1539928018 title "Chromatin modification contributes to senescence associated proliferation arrest" @default.
- W1539928018 hasPublicationYear "2012" @default.
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