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- W1541816652 endingPage "1756" @default.
- W1541816652 startingPage "1746" @default.
- W1541816652 abstract "Apoptosis is a critical process that intrinsically links organism survival to its ability to induce controlled death. Thus, functional apoptosis allows organisms to remove perceived threats to their survival by targeting those cells that it determines pose a direct risk. Central to this process are apoptotic caspases, enzymes that form a signalling cascade, converting danger signals via initiator caspases into activation of the executioner caspase, caspase-3. This enzyme begins disassembly of the cell by activating DNA degrading enzymes and degrading the cellular architecture. Interaction of pathogenic bacteria with caspases, and in particular, caspase-3, can therefore impact both host cell and bacterial survival. With roles outside cell death such as cell differentiation, control of signalling pathways and immunomodulation also being described for caspase-3, bacterial interactions with caspase-3 may be of far more significance in infection than previously recognized. In this review, we highlight the ways in which bacterial pathogens have evolved to subvert caspase-3 both through effector proteins that directly interact with the enzyme or by modulating pathways that influence its activation and activity." @default.
- W1541816652 created "2016-06-24" @default.
- W1541816652 creator A5035164805 @default.
- W1541816652 creator A5068098793 @default.
- W1541816652 date "2014-10-30" @default.
- W1541816652 modified "2023-09-30" @default.
- W1541816652 title "Bacterial secreted effectors and caspase‐3 interactions" @default.
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- W1541816652 doi "https://doi.org/10.1111/cmi.12368" @default.
- W1541816652 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4257569" @default.
- W1541816652 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25262664" @default.
- W1541816652 hasPublicationYear "2014" @default.