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- W1543094722 abstract "While central administration of arginine vasopressin (VP) to the non-febrile rat at high doses can cause hypothermia, there is little evidence for a role for endogenous VP in normal thermoregulation. In contrast, VP arising from cell bodies in the bed nucleus of the stria terminalis and innervating the ventral septal areas and possibly the amygdala appears to be an endogenous antipyretic, i.e. a substance capable of reducing fever. As the synthesis of VP in bed nucleus neurons is dependent upon circulating androgens, female rats have much less VP in these cells and their projections than do male rats. In keeping with this, females may make use of VP to a lessor extent than do males to bring about antipyresis. The phenomenon whereby the VP receptor can become sensitized by previous exposure to VP may be responsible for some states of endogenous antipyresis, in which fevers are suppressed through overactivity of the vasopressinergic system. States of endogenous antipyresis can be revealed around the time of parturition in both the neonate and the mother." @default.
- W1543094722 created "2016-06-24" @default.
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- W1543094722 creator A5039858998 @default.
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- W1543094722 date "1999-01-01" @default.
- W1543094722 modified "2023-09-23" @default.
- W1543094722 title "Arginine vasopressin, fever and temperature regulation" @default.
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- W1543094722 doi "https://doi.org/10.1016/s0079-6123(08)61582-4" @default.
- W1543094722 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/10074801" @default.
- W1543094722 hasPublicationYear "1999" @default.
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