Matches in SemOpenAlex for { <https://semopenalex.org/work/W1543288655> ?p ?o ?g. }
- W1543288655 abstract "All conditions causing pulmonary hypertension (PH) are characterized by three major changes in the pulmonary vasculature: vasoconstriction, vascular remodeling, and thrombosis [1,2,3]. Vascular remodeling includes muscularization of normally non-muscular peripheral pulmo‐ nary arteries, increase in medial wall thickness of muscular arteries, and increase in vascular connective tissue such as collagen and elastin [1,2,3]. Imbalance of vasoconstrictive and vasodilatory mediators might explain the increased vascular tone [1,2,3]. Endothelial cells synthesize and release prostacylin and nitric oxide for vasodilation as well as endothelin and thromboxane for vasoconstriction. Approved treatments for pulmonary arterial hypertension (PAH) include prostacyclins, endothelin receptor blockers, and phosphodiesterase-5 inhibitors as well as inhaled NO for persistent pulmonary hypertension of the neonate (PPHN) [2]." @default.
- W1543288655 created "2016-06-24" @default.
- W1543288655 creator A5012631564 @default.
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- W1543288655 creator A5033687211 @default.
- W1543288655 creator A5053170866 @default.
- W1543288655 creator A5091870290 @default.
- W1543288655 date "2013-07-17" @default.
- W1543288655 modified "2023-10-01" @default.
- W1543288655 title "Nitric Oxide in Pathophysiology and Treatment of Pulmonary Hypertension" @default.
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