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- W1547829603 abstract "Defective cellular-immune responses are characteristic of NZB/NZW mice and are important in the pathogenesis of their lupus-like immune-complex nephritis. T-cell function was further suppressed by the administration of antithymocyte globulin (ATG) to young female mice; controls received rabbit IgG or no treatment. Administration of ATG resulted in decreased circulating lymphocytes and diminished ability of spleen cells to induce graft-versus-host reactions and to function as helper or suppressor cells in antibody formation. ATG treatment accelerated proteinuria and deaths from nephritis and increased circulating antibodies to sDNA and nDNA. Renal eluates from ATG-treated mice contained increased quantities of mouse γ-globulin and anti-DNA antibodies, but antibodies to ATG were not demonstrable. Levels of serum neutralizing activity and antibodies against xenotropic and ecotropic murine C type viruses were not altered. The administration of ATG probably accelerated anti-DNA antibody formation and immune nephritis by reducing T-cell suppression of autoantibody formation." @default.
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- W1547829603 date "1977-09-01" @default.
- W1547829603 modified "2023-09-24" @default.
- W1547829603 title "The effect of altered lymphocyte function on the immunologic disorders of NZB/NZW mice" @default.
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- W1547829603 doi "https://doi.org/10.1016/0090-1229(77)90112-x" @default.
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