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• W1552990534 abstract "The effects of opiate receptor antagonism on the fetal cardiovascular response to hypoxemia were examined by means of the radionuclide-labeled microsphere technique. Heart rate, blood pressure, and cardiac output were measured during baseline perods, during hypoxemia, and before and after infusion of either naloxone (1 mg/kg) or an equivalent volume of 0.9% saline solution. Seventeen fetal sheep were subjected to maternal hypoxemia by allowing the ewes to breathe 10% oxygen (3% carbon dioxide, 87% nitrogen). The fetuses responded with bradycardia (p < 0.002 compared with control), increased blood pressure (p < 0.002 compared with control), and no significant change in combined ventricular output or placental blood flow. After naloxone, the bradycardia increased by 10% (p < 0.001), and both combined ventricular outout and placental blood flow fell by 20% (p < 0.01 and p < 0.01, respectively). The fetal bradycardic response to naloxone was reversible with atropine. In fetuses with normal oxygenation of the blood (normoxemic), naloxone had no significant effect on heart rate and blood pressure. These data indicate that endogenous opiates (e.g., endorphin and enkephalin) are important in regulating the fetal circulation during hypoxia, and that the effects of opiate receptor antagonism may be mediated through the autonomic nervous system." @default.
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• W1552990534 date "1982-08-01" @default.
• W1552990534 modified "2023-10-16" @default.
• W1552990534 title "Effects of naloxone on fetal circulatory responses to hypoxemia" @default.
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• W1552990534 doi "https://doi.org/10.1016/0002-9378(82)90477-x" @default.
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