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• W1553134728 abstract "Abstract For several decades, preceding the present, pathologists had observed the occasional occurrence of a tumor of a parathyroid gland in patients with generalized changes in the skeleton, characterized by dimineralization, fibrocystic disease and giant cell tumors, but considered the association merely coincidental. The relationship of these osseous changes to the tumors did not become evident until the relation of parathyroid function to calcium metabolism became known. There then followed an attempt to attribute various types of disease involving the skeleton such as osteitis deformens or Paget's disease, arthritis deformens, fragilitas ossea and localized fibrocystic disease to hyperfunction of the parathyroid glands. From both clinical and experimental data it has become recognized that hyperparathyroidism is a definite clinical entity characterized by a demonstrable disturbance of calcium metabolism. There is a generalized withdrawal of calcium from the skeleton which in many instances results in certain secondary changes designated as fibrocystic disease and giant cell tumors. This abnormal withdrawal results in hypercalcinemia, hypercalcinuria, hyperphosphouria and hypophosphonemia. Calcium outgo exceeds calcium intake referred to as a negative imbalance of calcium metabolism. Only in the presence of such evidences of a negative imbalance are we Justified in making a diagnosis of hyperparathyroidism." @default.
• W1553134728 created "2016-06-24" @default.
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• W1553134728 date "1937-07-01" @default.
• W1553134728 modified "2023-09-27" @default.
• W1553134728 title "Hyperparathyroidism" @default.
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• W1553134728 doi "https://doi.org/10.1016/s0002-9610(37)90865-1" @default.
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