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- W1555710735 endingPage "122" @default.
- W1555710735 startingPage "122" @default.
- W1555710735 abstract "During pregnancy there is an alteration in maternal immunity within the uterus where innate, proinflammatory immune responses are tightly regulated to prevent immunological rejection of the fetal allograft. Disruption of the delicate balance of cytokines by bacteria or other factors increases the production of proinflammatory cytokines at the maternal-fetal interface and activates the parturition mechanism prematurely. Despite years of searching, there is still no broadly effective strategy for preventing preterm labor and most therapies are directed at inhibiting myometrial contractions and improving neonatal outcome. Recent studies with progestins and interleukin-10 (IL-10), however, are showing promise in randomized clinical trials and animal studies. Furthermore, the identification of the Toll-like receptors as upstream mediators of inflammation may offer alternative therapeutic targets for preventing this common pregnancy complication." @default.
- W1555710735 created "2016-06-24" @default.
- W1555710735 creator A5082013157 @default.
- W1555710735 date "2003-01-01" @default.
- W1555710735 modified "2023-10-14" @default.
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