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- W1560744551 abstract "NEUROSCIENCEThe normal role of amyloid precursor protein (APP)--the source of amyloid b, the small protein whose abnormal deposition in the brain is thought to cause Alzheimer's disease--has remained elusive, despite years of study. Now, on page [115][1], neurobiologists report evidence that APP's so-called cytoplasmic tail, when released from APP, can activate gene expression in the nucleus--possibly acting directly as a transcription factor, a protein that binds to regulatory regions and turns genes on and off. This is the first time anyone has linked APP to control of gene expression. [1]: http://www.sciencemag.org/cgi/content/short/293/5527/115" @default.
- W1560744551 created "2016-06-24" @default.
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- W1560744551 date "2001-07-06" @default.
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- W1560744551 title "NEUROSCIENCE: Elusive Protein Auditions for Several New Roles" @default.
- W1560744551 doi "https://doi.org/10.1126/science.293.5527.28" @default.
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