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• W1561400456 abstract "The effects of short-term hypoprolactinemia on the pituitary-gonadal axis were evaluated in a group of patients with untreated prostatic carcinoma. Each patient was studied prior to and during 7-day oral administrations of bromocriptine (2.5 mg q.i.d.). Serum LH, prolactin (PRL), androst-4-ene-3,17 dione (androstenedione), testosterone, and 5α-androstane-3α,17β-diol (5α-Diol) levels, as well as intra-testicular testosterone, dihydrotestosterone (DHT), 5α-Diol and zinc (Zn) concentrations, were determined. Daily administration of bromocriptine caused a marked supression of serum PRL (mean ± SEM, 23.8 ± 2.5 vs. 6.4 ± 1.0 ng/ml) without concomitant changes in serum LH levels (mean ± SEM, 8.3 ± 1.6 vs. 8.9 ± 2.1 ng/ml). Hypoprolactinemia induced a significant decrease (P < 0.05) in the mean peripheral testosterone levels; but 5α-Diol and androstenedione remained unchanged. However, in testicular tissues, bromocriptine treatment resulted in significant increases in mean concentrations of total androgens (P < 0.001), testosterone (P < 0.001) and DHT (P < 0.02). Testicular levels of 5α-Diol were not significantly altered. There was no change in Zn levels in basal conditions and during bromocriptine administration. These results indicate that short-term suppression of serum PRL levels in man affects basal testicular function without altering serum LH. However, a direct action of bromocriptine on the human gonad cannot be excluded." @default.
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• W1561400456 date "1985-01-02" @default.
• W1561400456 modified "2023-09-27" @default.
• W1561400456 title "Induced Hypoprolactinemia and Testicular Steroidogenesis in Man" @default.
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• W1561400456 doi "https://doi.org/10.1002/j.1939-4640.1985.tb00811.x" @default.
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