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- W1562991209 abstract "Bone morphogenetic protein-4 (BMP-4), a member of the transforming growth factor-Beta(TGF-Beta) family, is overexpressed in different prolactinoma models and induces the development of these lineage adenomas. SMAD proteins activated by growth factors of the TGF-Beta and BMP family interact with estrogen receptors to stimulate the proliferation of prolactin and growth hormone-secreting cells. Furthermore, BMP-4 presents differential expression in normal and adenomatous corticotropes and inhibitory action on corticotropinoma cell proliferation. Moreover, BMP-4 mediates the antiproliferative action of retinoic acid in these cells. The present review highlights not only the crucial and opposite role of BMP-4 in the progression of pituitary adenomas but also that BMP-4 and retinoic acid interaction might serve as a potential new mechanism target for therapeutic approaches for Cushing disease." @default.
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- W1562991209 date "2006-01-01" @default.
- W1562991209 modified "2023-10-14" @default.
- W1562991209 title "Bone Morphogenetic Protein-4 Control of Pituitary Pathophysiology" @default.
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- W1562991209 doi "https://doi.org/10.1159/000094262" @default.
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