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- W156698508 abstract "Disruption of glutamate homeostasis is a contributing factor to brain injury in several pathological conditions including epilepsy. Excitatory amino acid transporters, or EAATs, are responsible for maintaining levels of extracellular glutamate below pathological levels in the human brain, offering a potential therapeutic target. Recent pharmacological investigations have revealed the structure of the ligand binding site on EAATs and suggest new directions for the design of transported and nontransported inhibitors. Advances in techniques for assessing protein structure have further expanded our understanding in this field. Unique targeting and trafficking pathways for EAATs play a critical role in EAAT dynamics and new advances in these technologies will further our understanding of glutamate homeostasis in the healthy and injured brain." @default.
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- W156698508 date "2009-01-01" @default.
- W156698508 modified "2023-09-24" @default.
- W156698508 title "TRANSPORTERS | Glutamate Transporters in Epilepsy" @default.
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- W156698508 doi "https://doi.org/10.1016/b978-012373961-2.00003-5" @default.
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