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- W1567184779 abstract "Publisher Summary The scientific study of epilepsy is done to expose an organism to an epileptogenic intervention, electrical, pharmacological, and genetic etcetera manipulate the parameters to determine how these influence epileptogenicity, and hence draw inferences, concerning the underlying mechanisms. In the reflex epilepsies graded responses can be elicited by stimuli of gradually increasing epileptogenicity, ranging from subclinical electroencephalography (EEG) discharges through minor, to more severe ictal events. By using stimuli that are never more than marginally supraliminal, ethically acceptable experimental studies of human epileptogenesis can, thus, be performed with minimal risk of inducing a convulsive seizure. Given the unpredictability, reflex epilepsies provide an unusual opportunity for controlled activation of the epileptogenic process. There is discussion on photosensitivity as an experimental model. In human photosensitivity, the epileptogenic process is initiated in parieto-occipital cortex. The conditions required to establish a discharge can be inferred from the stimulus characteristics that determine epileptogenicity. The experimental procedure employed in most of the work cited here involve potentially presenting epileptogenic visual stimuli to susceptible subjects and manipulating a single stimulus characteristic to determine the threshold value for eliciting a discharge and the relationship to discharge probability. The factors on which the pattern of a typical generalized photoparoxysmal response depends are discussed in the chapter. These findings imply that the establishment of an epileptic discharge depends on adequate, synchronous activation of a sufficient population of functionally related neurones. Somatosensory induced seizures are seen in patients with unilateral structural abnormalities in the somatosensory area and the seizures are appropriately lateralized and presumably reflect cortical hyperexcitability in the sensory cortex. Cognitive triggering is mainly seen in idiopathic generalized epilepsy. Epileptogenesis involves both hyperactivity and hypersynchrony and sensory stimuli can both activate and desynchronize neuronal activity. Facilitation or inhibition of seizure activity may depend on the balance between these processes." @default.
- W1567184779 created "2016-06-24" @default.
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- W1567184779 date "2002-01-01" @default.
- W1567184779 modified "2023-09-27" @default.
- W1567184779 title "Chapter 69 Reflex epilepsy" @default.
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- W1567184779 doi "https://doi.org/10.1016/s1567-424x(09)70489-1" @default.
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