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- W1569579747 abstract "Diabetic nephropathy (DN) is one of the complications of diabetes. The presence of high serum glucose concentrations can cause an irreversible progression of renal dysfunction, including glomerulosclerosis, arteriolar hyalinosis, and tubulointerstitial fibrosis. Oxidative stress has been linked to the pathogenesis of DN. The major sources of reactive oxygen species (ROS) in kidney are induced by the polyl pathway, nicotinamide adenine dinucleotide phosphate (NADPH) oxidases, protein kinase C (PKC), and advanced glycation end products (AGEs). A huge increase in ROS can cause protein denaturation and cell death by changing protein sensors or intracellular signaling pathways. In addition to the traditional clinical strategies of controlling glucose and dietary protein, providing antihypertensive treatment, and reducing hyperlipidemia, there are several new approaches to the treatment of diabetic nephropathy that are based on decreasing ROS. Here we review recent research describing the relationship between oxidative stress and DN, and the application of antioxidants to the treatment of DN." @default.
- W1569579747 created "2016-06-24" @default.
- W1569579747 creator A5017953898 @default.
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- W1569579747 date "2013-06-01" @default.
- W1569579747 modified "2023-10-18" @default.
- W1569579747 title "Oxidative Stress in Diabetic Nephropathy" @default.
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