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- W1570288009 abstract "textabstractThe particular association of RA with anti-lgG antibodies suggests an important roleof lgG in the etiology and pathology of RA. One of the suggested mechanisms bywhich lgG could be altered is exposure to oxygen free radicals. During inflammationlarge amounts of oxygen free radicals are produced by PMN giving basis to thissuggestion. In Chapter II of this thesis a general overview is presented on the role ofoxygen free radicals in RA. Particular attention is given to the effects on proteins. Fromthis it can be seen that oxygen free radicals play an important role in the inflammatoryresponse and concomitant tissue damage in RA.Briefly, the attention in this thesis is focussed on the following questions:- Can lgG be altered by oxygen free radicals?- What is the nature of these alterations?- Does oxygen free radical altered lgG interact with rheumatoid factors andcomplement?- What is the effect of oxygen free radical altered lgG on the non-specific immunesystem (granulocytes)?- What is the effect of oxygen free radical altered lgG on the specific immunesystem (lymphocytes)?" @default.
- W1570288009 created "2016-06-24" @default.
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- W1570288009 date "1990-05-02" @default.
- W1570288009 modified "2023-09-26" @default.
- W1570288009 title "Oxygen free radical altered immunoglobin G in the etiopathogenesis of rheumatoid arthritis" @default.
- W1570288009 hasPublicationYear "1990" @default.
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