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• W1570479580 startingPage "496" @default.
• W1570479580 abstract "Hormones, neurotransmitters, and growth factors initiate cyto-solic Ca2+ (Cai 2+) signaling through a variety of mechanisms. As observations move from cell populations to single cells to subcellular regions, the complexity of Cai 2+ signaling patterns increases and the time scale of signaling events decreases. Cai 2+ signaling has been examined to a lesser extent in bile duct epithelia than in hepatocytes. Ca2+ signaling also plays a role in lipid metabolism and the pathogenesis of non-alcoholic fatty liver disease. Ca2+ regulates a wide range of functions in liver. Early studies in isolated perfused rat liver showed that the net effect of Cai 2+ on bile acid-independent bile flow is inhibitory, but more recent studies demonstrate that both bile acid-independent and bile acid-dependent flow are potentiated by Ca2+ signaling in hepatocytes. Ca2+ signals are associated with progression through the cell cycle. Cai 2+ directly regulates fluid and electrolyte secretion in bile duct epithelia." @default.
• W1570479580 created "2016-06-24" @default.
• W1570479580 creator A5009410103 @default.
• W1570479580 creator A5036843004 @default.
• W1570479580 creator A5064754617 @default.
• W1570479580 date "2020-01-24" @default.
• W1570479580 modified "2023-10-17" @default.
• W1570479580 title "Ca²⁺Signaling in the Liver" @default.
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