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- W1570642610 abstract "Embryonic stem (ES) cells isolated from the inner cell mass (ICM) of blastocysts possess the defining pluroptency: unlimited self-renewal and giving rise to all cells of the organ‐ ism[1, 2]. Thus, ES cells hold great promise for regenerative medicine to treat many dis‐ eases including heart failure, diabetes, Alzheimer’s and Parkinson’s disease by replacing the damaged cells with ES cell-derived healthy ones. The recent advent of induced pluri‐ potent stem (iPS) cells reprogrammed from somatic cells has the potential to revolution‐ ize the field of regenerative medicine since patient-derived iPS cells, in principle, circumvent the ethical problems and immune rejection associated with human ES cells[3]. Nevertheless, the future clinical translation of ES cells and iPS cells is facing nu‐ merous hurdles. Understanding the molecular mechanisms that impart ES cells with plu‐ ripotency may help address some of these challenges. The past few years have seen tremendous progress in understanding of mechanisms which govern ES cell pluripoten‐ cy. In this chapter, we will review critical signaling and transcription factor networks that have been identified to maintain ES cell pluripotency." @default.
- W1570642610 created "2016-06-24" @default.
- W1570642610 creator A5046138088 @default.
- W1570642610 creator A5070757563 @default.
- W1570642610 creator A5083228477 @default.
- W1570642610 date "2013-08-28" @default.
- W1570642610 modified "2023-09-27" @default.
- W1570642610 title "Molecular Mechanisms of Embryonic Stem Cell Pluripotency" @default.
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