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• W1570798316 abstract "Significance An attribute of adaptive immunity is the generation of memory cells that mount enhanced responses after rechallenge. While memory is best understood for αβT cell receptor-bearing lymphocytes, the properties of γδT cells in the context of a secondary challenge have been less examined. After murine skin inflammation with imiquimod, IL-17–secreting γδT cells expand in lymph nodes and traffic to skin where they persist as memory-like cells capable of rapid activation upon rechallenge. This enhanced secondary response is in part mediated by increased IL-1R1 expression. Expansion of potentially pathogenic memory cells in lymph nodes and redistribution throughout normal and inflamed skin may help explain the generalized worsening of psoriasis reported in patients undergoing localized skin treatment with imiquimod." @default.
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• W1570798316 date "2015-06-15" @default.
• W1570798316 modified "2023-10-16" @default.
• W1570798316 title "Inflammation induces dermal Vγ4 ⁺ γδT17 memory-like cells that travel to distant skin and accelerate secondary IL-17–driven responses" @default.
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• W1570798316 doi "https://doi.org/10.1073/pnas.1508990112" @default.
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