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- W157098193 abstract "The transmission of pain from the periphery through to the higher centres of the brain is no longer viewed as a passive process of impulses in exclusive pathways. The final sensation of pain results from messages arising from the interplay between neuronal systems, both excitatory and inhibitory, at many levels of the central nervous system but which converge markedly on the spinal cord. It has long been recognized from human experience and clinical data that there is not always a clear relation between the stimulus and the response to pain in an individual — hyperalgesia, allodynia (touch evoked pain) and prolonged painful sensations can occur in some circumstances whilst in other situations, injury is not always accompanied by pain. This plasticity, the ability for transmission in nociceptive systems to change, can result from changes over short time courses. Peripheral inflammation provides an excellent example of how changes in the periphery can markedly alter the ways in which the central nervous system responds to a given stimulus and, in particular, the extent to which the pharmacology of transmitter systems in the spinal cord are subject to plasticity [1–5]." @default.
- W157098193 created "2016-06-24" @default.
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- W157098193 date "1999-01-01" @default.
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- W157098193 title "The roles of spinal receptors in nociceptive responses" @default.
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- W157098193 doi "https://doi.org/10.1007/978-3-0348-8753-3_2" @default.
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