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- W1574220082 endingPage "80" @default.
- W1574220082 startingPage "71" @default.
- W1574220082 abstract "Rac and PI3Ks are intracellular signal transducers able to regulate multiple signaling pathways fundamental for cell behavior. PI3Ks are lipid kinases that produce phosphorylated lipids which, in turn, transduce extracellular cues within the cell, while Rac is a small G protein that impacts on actin organization. Compelling evidence indicates that in multiple circumstances the 2 signaling pathways appear intermingled. For instance, phosphorylated lipids produced by PI3Ks recruit and activate GEF and GAP proteins, key modulators of Rac function. Conversely, PI3Ks interact with activated Rac, leading to Rac signaling amplification. This review summarizes the molecular mechanisms underlying the cross-talk between Rac and PI3K signaling in 2 different processes, cell migration and ROS production." @default.
- W1574220082 created "2016-06-24" @default.
- W1574220082 creator A5022866518 @default.
- W1574220082 creator A5038307963 @default.
- W1574220082 creator A5045008462 @default.
- W1574220082 creator A5050851929 @default.
- W1574220082 creator A5062527845 @default.
- W1574220082 date "2015-04-03" @default.
- W1574220082 modified "2023-10-17" @default.
- W1574220082 title "Crossroads of PI3K and Rac pathways" @default.
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