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- W158267923 abstract "The classification of secondary cardiomyopathies is based on “myocardial damage within the scope of a systemic disease.” In view of this definition, myocardial impairment in sepsis and the multiple organ dysfunction syndrome might be reckoned as a secondary form of cardiomyopathy, as septic cardiomyopathy [1]: various infectious disease entities and myocardial virulence factors may trigger common mediator pathways. Thereby, a complex pattern of myocardial depression with different grades of severity may result, characterized on the one hand by common principles (e.g., catecholamine desensitization and the effects of tumor necrosis factor α (TNFα); see below), and on the other hand by toxin-specific and therefore infectious agent-specific pathways (e.g., inhibition of protein synthesis by P. exotoxin A; see below). In a canine model of endotoxin shock, development of myocardial dysfunction indicates a bad prognosis [2]. In patients, heart failure can be documented to a similar degree in various forms of Gram-negative and Gram-positive sepsis and also in fungal sepsis (figure 1)." @default.
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- W158267923 date "1996-01-01" @default.
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- W158267923 title "Impaired Cellular Signaling of the Adenylyl Cyclase and the Phosphoinositide Pathway in Septic Cardiomyopathy" @default.
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- W158267923 doi "https://doi.org/10.1007/978-1-4613-1235-2_18" @default.
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