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• W15881265 abstract "Survival in B-cell chronic lymphocytic leukemia (CLL) is poor if one of the following risk factors is present: advanced stage, short lymphocyte doubling time, unfavorable cytogenetics, CD38 marker, or unmutated status of the variable region of the immunoglobulin heavy chain gene (1–4). The use of intensive myeloablative therapy followed by autologous or allogeneic stem cell transplantation (SCT) for CLL has greatly increased over the last few years (5–13). As the safety and efficacy of autotransplantation rely on supportive care, improvements in peripheral blood stem cell (PBSC) mobilization and the cytotoxic therapy administered can induce long-lasting clinical and molecular remissions (8,9,13–15). The mortality of the procedure could be further reduced and should be well below 10% (8,9),but recently, in most series, a continuous increase in molecular relapses has been observed after autotransplantation. In allogeneic settings, the graft-vs-leukemia (GVL) activity should be responsible for very substantial disease control (16,17) and seems to cure a subset of poor-risk patients but at the price of an excessively high treatment-related mortality (TRM) (13). The recent development of a nonmyeloablative conditioning regimen followed by donor lymphocyte infusion (DLI) may help to improve the tolerability of allogeneic SCT in patients with CLL (18–27). Nevertheless, several issues in SCT for CLL remain unsettled, and all data concerning this settings are important in an attempt to build a therapeutic strategy for CLL including conventional chemotherapy, serotherapy, autotransplantation, and a standard or reduced-intensity conditioning regimen followed by a related or unrelated allotransplantation." @default.
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• W15881265 date "2004-01-01" @default.
• W15881265 modified "2023-09-27" @default.
• W15881265 title "Autologous and Allogeneic Transplantations for Chronic Lymphocytic Leukemia" @default.
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• W15881265 doi "https://doi.org/10.1007/978-1-59259-412-2_14" @default.
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