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- W1591420606 abstract "Analyses of transgene silencing phenomena in plants and other organisms have revealed the existence of epigenetic silencing mechanisms that are based on recognition of nucleic acid sequence homology at either the DNA or RNA level. Common triggers of homology-dependent gene silencing include inverted DNA repeats and double-stranded RNA, a versatile silencing molecule that can induce both degradation of homologous RNA in the cytoplasm and methylation of homologous DNA sequences in the nucleus. Inverted repeats might be frequently associated with silencing because they can potentially interact in cis and in trans to trigger DNA methylation via homologous DNA pairing, or they can be transcribed to produce double-stranded RNA. Homology-dependent gene silencing mechanisms are ideally suited for countering natural parasitic sequences such as transposable elements and viruses, which are usually present in multiple copies and/or produce double-stranded RNA during replication. These silencing mechanisms can thus be regarded as host defense strategies to foreign or invasive nucleic acids. The high content of transposable elements and, in some cases, endogenous viruses in many plant genomes suggests that host defenses do not always prevail over invasive sequences. During evolution, slightly faulty genome defense responses probably allowed transposable elements and viral sequences to accumulate gradually in host chromosomes and to invade host genes. Possible beneficial consequences of this foreign DNA buildup include the establishment of genome defense-derived epigenetic control mechanisms for regulating host gene expression and acquired hereditary immunity to some viruses." @default.
- W1591420606 created "2016-06-24" @default.
- W1591420606 creator A5031570460 @default.
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- W1591420606 creator A5069041693 @default.
- W1591420606 creator A5080264876 @default.
- W1591420606 creator A5080392675 @default.
- W1591420606 date "2002-01-01" @default.
- W1591420606 modified "2023-10-06" @default.
- W1591420606 title "8 Homology-dependent gene silencing and host defense in plants" @default.
- W1591420606 cites W101038835 @default.
- W1591420606 cites W1493912125 @default.
- W1591420606 cites W1496670813 @default.
- W1591420606 cites W1500148822 @default.
- W1591420606 cites W1500702901 @default.
- W1591420606 cites W1533942477 @default.
- W1591420606 cites W1534256444 @default.
- W1591420606 cites W1538689496 @default.
- W1591420606 cites W1546331692 @default.
- W1591420606 cites W1550720320 @default.
- W1591420606 cites W1552456601 @default.
- W1591420606 cites W1555644224 @default.
- W1591420606 cites W1556385076 @default.
- W1591420606 cites W1557790541 @default.
- W1591420606 cites W1563581981 @default.
- W1591420606 cites W1564420027 @default.
- W1591420606 cites W1573631089 @default.
- W1591420606 cites W1588101546 @default.
- W1591420606 cites W1594381468 @default.
- W1591420606 cites W1602141296 @default.
- W1591420606 cites W1606677480 @default.
- W1591420606 cites W1643938048 @default.
- W1591420606 cites W1647075334 @default.
- W1591420606 cites W1816016449 @default.
- W1591420606 cites W1838266191 @default.
- W1591420606 cites W1889938952 @default.
- W1591420606 cites W1916132949 @default.
- W1591420606 cites W1916491745 @default.
- W1591420606 cites W1921593493 @default.
- W1591420606 cites W1925002711 @default.
- W1591420606 cites W1932753289 @default.
- W1591420606 cites W1933986198 @default.
- W1591420606 cites W1936021374 @default.
- W1591420606 cites W1960783150 @default.
- W1591420606 cites W1963759242 @default.
- W1591420606 cites W1967159396 @default.
- W1591420606 cites W1970535781 @default.
- W1591420606 cites W1976208541 @default.
- W1591420606 cites W1976346232 @default.
- W1591420606 cites W1976601441 @default.
- W1591420606 cites W1978523653 @default.
- W1591420606 cites W1978627551 @default.
- W1591420606 cites W1979404059 @default.
- W1591420606 cites W1979965542 @default.
- W1591420606 cites W1984155772 @default.
- W1591420606 cites W1986167448 @default.
- W1591420606 cites W1989837887 @default.
- W1591420606 cites W1993078372 @default.
- W1591420606 cites W1994157469 @default.
- W1591420606 cites W1995679130 @default.
- W1591420606 cites W2001835861 @default.
- W1591420606 cites W2003520310 @default.
- W1591420606 cites W2004722288 @default.
- W1591420606 cites W2006100734 @default.
- W1591420606 cites W2006101755 @default.
- W1591420606 cites W2008572767 @default.
- W1591420606 cites W2012299307 @default.
- W1591420606 cites W2017373213 @default.
- W1591420606 cites W2018220730 @default.
- W1591420606 cites W2018651477 @default.
- W1591420606 cites W2020850118 @default.
- W1591420606 cites W2023000785 @default.
- W1591420606 cites W2025797510 @default.
- W1591420606 cites W2026556151 @default.
- W1591420606 cites W2028601289 @default.
- W1591420606 cites W2030295209 @default.
- W1591420606 cites W2031372024 @default.
- W1591420606 cites W2031950838 @default.
- W1591420606 cites W2033285403 @default.
- W1591420606 cites W2036300762 @default.
- W1591420606 cites W2037002709 @default.
- W1591420606 cites W2037593765 @default.
- W1591420606 cites W2041944855 @default.
- W1591420606 cites W2044023120 @default.
- W1591420606 cites W2044543540 @default.
- W1591420606 cites W2044588477 @default.
- W1591420606 cites W2045450761 @default.
- W1591420606 cites W2046241907 @default.
- W1591420606 cites W2052639867 @default.
- W1591420606 cites W2054383888 @default.
- W1591420606 cites W2056156549 @default.
- W1591420606 cites W2057132428 @default.
- W1591420606 cites W2060309032 @default.
- W1591420606 cites W2060446375 @default.
- W1591420606 cites W2062188301 @default.
- W1591420606 cites W2062916004 @default.
- W1591420606 cites W2064745039 @default.
- W1591420606 cites W2070792578 @default.