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• W159308567 abstract "Late damage to normal tissues is loosely defined as pathological changes in tissue systems that occur months to years after delivery of the radiation dose. In some cases, consideration is given to tissue effects that occur within weeks after irradiation. The more general definition of late effects describes any untoward changes in organs and tissues after irradiation, and often in tissues or organs not irradiated for effect. The doses required to elicit late responses frequently may be larger than those associated with most of the early, acute responses, and for this reason, they are most often encountered with fractionated regional radiation of the type administered in radiotherapy for malignant or other diseases. The fractionation protects organs from acute effects, but the late changes are less modified. The animals or patients would not normally survive total body radiation doses at the dose levels usually associated with the onset of effects in these late responding tissues. Two approaches to explanations of the underlying cause of late effects from ionizing radiation have received a great deal of recent attention. One group emphasizes cellular depletion (target cell depletion) of clonogenic elements or functional elements that require replacement from clonogenic pools. Another group suggests that the central process underlying all late effects in organ systems damages the vascular endothelium, which leads to vascular insufficiency." @default.
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• W159308567 date "1998-01-01" @default.
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• W159308567 title "Late Effects of Radiation on Normal Tissues: Nonstochastic Effects" @default.
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• W159308567 doi "https://doi.org/10.1016/b978-012053085-4/50013-2" @default.
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