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- W1596601119 endingPage "1493" @default.
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- W1596601119 abstract "One of the mechanisms by which the experience-dependent reorganization of neural circuitry can occur is through changes in synaptic strength. Almost every excitatory synapse in the mammalian brain exhibits LTP (long-term potentiation) or LTD (long-term depression), two cellular mechanisms of synaptic plasticity. However, LTP and LTD have been reported much more rarely at fast inhibitory GABA(A) receptor synapses. Our recent study suggests that in vivo morphine initiates a long-lasting alteration of GABAergic synapses in the ventral tegmental area (VTA) by blocking the mechanisms required for LTP of GABAergic synapses. Here we put this work into the context of other examples of synaptic plasticity at GABAergic synapses." @default.
- W1596601119 created "2016-06-24" @default.
- W1596601119 creator A5048550774 @default.
- W1596601119 creator A5085437683 @default.
- W1596601119 date "2008-03-15" @default.
- W1596601119 modified "2023-10-14" @default.
- W1596601119 title "LTP of GABAergic synapses in the ventral tegmental area and beyond" @default.
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- W1596601119 doi "https://doi.org/10.1113/jphysiol.2007.148098" @default.
- W1596601119 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2375707" @default.
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