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- W160005519 abstract "The endocrine cells of the pancreatic islet organ respond very effectively to the blood’s small nutrient molecules: Glucose and the mixture of 20 standard amino acids are potent stimulants at physiological concentrations, and fatty acids and ketone bodies also appear to be effective. The high efficacy of these molecules contrasts with the inertness of major metabolites, as for instance pyruvate, lactate, glycerol, uric acid, urea, and ammonia, which have no influence on these cells at physiological concentrations. The examination of this issue is naturally greatly influenced by the prevailing views about the biochemical nature of fuel sensing, in particular the essential role that intermediary metabolism plays in this process () as contrasting to concepts of fuel sensing mechanisms that might be based on direct fuel interactions with membrane receptors and ion channels (), circumventing catabolism of the agonists. It is currently widely accepted that the effect of most nutrient agonists on pancreatic endocrine cells is a result of their catabolism (). It is the purpose of this brief review to provide a perspective on this topic." @default.
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- W160005519 date "1994-01-01" @default.
- W160005519 modified "2023-09-23" @default.
- W160005519 title "Fundamentals of Fuel Sensing and Intermediary Metabolism in Pancreatic A-and B-Cells" @default.
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- W160005519 doi "https://doi.org/10.1007/978-1-4612-0241-7_8" @default.
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