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• W1602706680 abstract "High anti-DNP PFC responses to DNP-DE or DNP-KLH were obtained by transferring normal or primed FcR− B cell fractions into irradiated syngeneic recipients. On the other hand, the FcR+ B cell fraction showed a low precursor activity. Trypsinization of the FcR+ B cells, to eliminate remaining antigen-antibody complexes on the surface, failed to augment the response in comparison with that of trypsin-untreated FcR+ B cells. Therefore, the weak precursor activity of FcR+ B cells seemed to be inherent. No synergistic interaction between the FcR+ B and precursor FcR− B cells, to give rise to the maximum PFC response, was observed. On the contrary, the FcR+ B cells significantly suppressed the PFC responses of FcR− B cells. This kind of suppression could be mediated by a factor released from the FcR+ B cell, but not from the FcR− B or original-unrosetted spleen cell fraction. The factor was not attributable to macrophages, because the FcR+ B cells isolated from normal spleen cells, of which macrophages were depleted by Sephadex G-10 columns, could produce the factor with the same activity. Stimulation by specific antigen is not necessary for the induction of the factor(s) as well as of the suppressing FcR+ B cells. It seems to be necessary to stimulate FcR by antigen-antibody complexes to produce or release this factor." @default.
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• W1602706680 date "1978-09-01" @default.
• W1602706680 modified "2023-09-27" @default.
• W1602706680 title "Immunological properties of Fc receptor on lymphocytes" @default.
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• W1602706680 doi "https://doi.org/10.1016/0008-8749(78)90100-4" @default.
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