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- W1606396102 endingPage "923" @default.
- W1606396102 startingPage "906" @default.
- W1606396102 abstract "There is growing evidence that altered production and/or spatio‐temporal distribution of reactive oxidant species and reactive nitrosative species in blood creates oxidative and/or nitrosative stresses in the failing myocardium and endothelium. This contributes to the abnormal cardiac and vascular phenotypes that characterize cardiovascular disease. These derangements at the system level can now be interpreted at the integrated cellular and molecular levels in terms of effects on signaling elements in the heart and vasculature. The end results of nitric oxide/redox disequilibrium have implications for cardiac and vascular homeostasis and may result in the development of atherosclerosis, myocardial tissue remodelling and hypertrophy. Reactive oxygen species/reactive nitrogen species generation is also attributed to the transit from hypertrophic to apoptotic phenotypes, a possible mechanism of myocardial failure. In this review, we highlight the possible roles of altered production and/or spatio‐temporal distribution of reactive oxidant species and reactive nitrosative species in blood on the pathogenesis of the failing cardiovascular system." @default.
- W1606396102 created "2016-06-24" @default.
- W1606396102 creator A5038771290 @default.
- W1606396102 creator A5073758619 @default.
- W1606396102 creator A5086090291 @default.
- W1606396102 date "2007-01-23" @default.
- W1606396102 modified "2023-10-18" @default.
- W1606396102 title "Nitric oxide in blood" @default.
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