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• W1612794657 abstract "The following four possible pathways for itching sensation have been suggested by recent reports. 1) Histaminergic TRPV1-positive pathway: Although histamine-positive nerve fibers cannot strictly be classified as itch specific due to their excitation also by pure algogens (making them itch-selective), the existence of a subpopulation of nociceptors responsible for itching is strongly suggested. Moreover, the TRPV1-expressing neurons have been suggested to be the main sensors and mediators of itching. 2) Histaminergic TRPV1-negative pathway: The scratching behavior caused by itching was not different between capsaicin-pre-treated and vehicle-treated (control) mast cell-rich NC mice. This result suggests the existence of a capsaicin-insensitive (TRPV1-negative) histaminergic pathway. 3) Non-histaminergic PAR-2 pathway: Protease-activated receptor 2 (PAR-2) has been shown to play a role in the itching of atopic dermatitis (AD). The itch evoked by cowhage (a non-histaminergic pruritogen that activates PAR-2) is very similar in characteristics to the itch evoked by conditions such as AD. 4) Non-histaminergic serotonin (5-HT) pathway: 5-HT alone applied to the human skin evokes an itching sensation and has been suggested to be involved in the itching associated with pruritic diseases, such as polycythemia vera and cholestasis." @default.
• W1612794657 created "2016-06-24" @default.
• W1612794657 creator A5031651644 @default.
• W1612794657 creator A5062434804 @default.
• W1612794657 date "2013-07-01" @default.
• W1612794657 modified "2023-09-23" @default.
• W1612794657 title "Four Possible Itching Pathways Related to the TRPV1 Channel, Histamine, PAR-2 and Serotonin." @default.
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• W1612794657 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3773347" @default.
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• W1612794657 hasPublicationYear "2013" @default.
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