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- W161858356 endingPage "461" @default.
- W161858356 startingPage "437" @default.
- W161858356 abstract "Publisher SummaryStudies suggest a dose-dependent relationship between alcohol consumption and fracture risk in both women and men. This increased risk may be attributable to a reduction in bone density in those with excessive alcohol intake. Alcoholic bone disease is characterized by impaired bone formation in the face of relatively normal bone resorption. The uncoupling of these two physiologic processes results in defective remodeling of skeletal tissue and, in turn, reduced bone mass and increased fracture risk. The growing skeleton may be especially sensitive to the adverse effects of alcohol. Experiments using well-defined osteoblastic model systems indicate that the observed reductions in bone formation result from a direct, antiproliferative effect of ethanol on the osteoblast itself. At present, sustained reduction in alcohol intake is the only effective therapy for alcohol-induced bone disease. Unfortunately, the long-term success of such therapy is poor. An improved understanding of the pathogenesis of alcohol-induced bone disease may lead to alternative therapeutic avenues." @default.
- W161858356 created "2016-06-24" @default.
- W161858356 creator A5012080821 @default.
- W161858356 date "1999-01-01" @default.
- W161858356 modified "2023-09-27" @default.
- W161858356 title "Alcohol" @default.
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