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- W162640051 abstract "Glutamic acid functions as excitatory neurotransmitter in mammalian central nervous systems. Hippocampus is characterized as most glutaminergic rich area in the cerebrum, which plays key role for many neural activities. Several naturally occuring epilepsy is related to the disorder of glutaminergic nervous systems. Kainic acid and N-methyl-Daspartic acid (NMDA) are well known agonists which bind different subclass of glutaminergic receptors. These agonists, therefore, have potent. neurotoxicity, of which example is that kainic acid-induced seizure is a good model for naturally occuring epilepsy. During the seizure, the marked increases in oxygen consumption and blood-flow are well-recognized and the latter overcomes the former, resulting the increase in hemoglobin oxygenation state. The redox behavior of intracellular space, mitochondria, is, however, not well characterized yet. Recently, the marked and transient reduction of cytochrome oxidase was demonstrated during bicucul-lin-induced seizue (1), where near-infrared spectrophotometry was used for simultaneous monitoring the hemoglobin oxygenation state and redox state of mitochondria. The redox state of copper not heme moeity of cytochrome oxidase was measured selectively by this method (2). In this paper using near-infrared photometry, we will report the changes of oxygenation states of brain fissure of rat during glutamate-agonists induced epileptic seizure." @default.
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- W162640051 date "1994-01-01" @default.
- W162640051 modified "2023-09-23" @default.
- W162640051 title "Changes in Oxygenation States of Rat Brain Tissues during Glutamate-Related Epileptic Seizures - Near-Infrared Study" @default.
- W162640051 cites W1796037372 @default.
- W162640051 doi "https://doi.org/10.1007/978-1-4615-2468-7_77" @default.
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