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• W1644992952 endingPage "7091" @default.
• W1644992952 startingPage "7085" @default.
• W1644992952 abstract "Abstract In recent years, several investigators have unraveled a previously unrecognized role for G-CSF in the regulation of T cell and dendritic cell functions. The experimental evidence in favor of G-CSF-mediated immune regulation includes the ability to switch T cell cytokine secretion profile to Th2 responses and the promotion of regulatory T cell and tolerogenic dendritic cell differentiation. Interestingly, G-CSF is beneficial in animals for the prevention and/or treatment of immune-mediated diseases, e.g., graft-vs-host disease, multiple sclerosis, systemic lupus erythematosus, inflammatory bowel disease, and diabetes, suggesting a potential role in human autoimmune diseases. This review summarizes the growing body of evidence that supports a critical role for G-CSF as a novel mediator of T cell tolerance." @default.
• W1644992952 created "2016-06-24" @default.
• W1644992952 creator A5016603192 @default.
• W1644992952 creator A5029407987 @default.
• W1644992952 creator A5054676984 @default.
• W1644992952 creator A5066649201 @default.
• W1644992952 creator A5067087473 @default.
• W1644992952 date "2005-12-01" @default.
• W1644992952 modified "2023-10-14" @default.
• W1644992952 title "Granulocyte Colony-Stimulating Factor: A Novel Mediator of T Cell Tolerance" @default.
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• W1644992952 doi "https://doi.org/10.4049/jimmunol.175.11.7085" @default.
• W1644992952 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16301609" @default.

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