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• W165035215 abstract "The in vivo functions of interleukin-3 (IL-3) were investigated by generating IL-3-deficient mice. Although hematopoiesis was unimpaired in homozygous mutant animals, contact hypersensitivity reactions were compromised. IL-3 was required for efficient priming of hapten-specific contact hypersensitivity responses, but was dispensable for T-cell-dependent sensitization to tumor cells. These findings reveal a critical role for IL-3 in some forms of delayed-type hypersensitivity." @default.
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• W165035215 date "1998-02-01" @default.
• W165035215 modified "2023-09-28" @default.
• W165035215 title "Involvement of Interleukin-3 in Delayed-Type Hypersensitivity" @default.
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• W165035215 doi "https://doi.org/10.1182/blood.v91.3.778" @default.
• W165035215 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/9446636" @default.
• W165035215 hasPublicationYear "1998" @default.
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