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- W165035876 abstract "For approximately fifty years, experimental allergic encephalomyelitis (EAE) has been widely studied. This disease was initially described by Rivers and co-workers (1) as a laboratory model for post-vaccinal encephalomyelitis (PVE). The production of EAE in these initial studies required multiple injections of whole spinal cord homogenate (SCH). After the introduction of Freund’s adjuvant, it became possible to produce EAE in some species with a single injection of SCH (2,3). This facilitated analysis of the responsible encephalitogen which was found to be a component of myelin and subsequently was identified as the myelin basic protein (MBP) (4). Because of pathological similarities between PVE and multiple sclerosis (MS), it has been suspected that similar mechanisms may be operative in the two diseases; however, traditionally EAE has been regarded as an imperfect model for MS because its course is usually monophasic. In 1965, Stone and Lerner (5) described a chronic form of the disease in guinea pigs, and in recent years there has been renewed interest in the investigation of relapsing EAE (REAE) because of its potential relevance to MS (6,7)." @default.
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- W165035876 date "1983-01-01" @default.
- W165035876 modified "2023-09-23" @default.
- W165035876 title "Murine Experimental Allergic Encephalomyelitis" @default.
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- W165035876 doi "https://doi.org/10.1007/978-3-642-69094-5_5" @default.
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